Effects of Neonatal Iron Feeding and Chronic Clioquinol Administration on the Parkinsonian Human A53T Transgenic Mouse
Author:
Affiliation:
1. The Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Parkville, Victoria 3052, Australia
2. Elemental Bio-imaging Facility, University of Technology Sydney, Broadway, New South Wales 2007, Australia
Funder
National Health and Medical Research Council
Australian Research Council
Publisher
American Chemical Society (ACS)
Subject
Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine
Link
http://pubs.acs.org/doi/pdf/10.1021/acschemneuro.5b00305
Reference34 articles.
1. A delicate balance: Iron metabolism and diseases of the brain
2. The role of iron in brain ageing and neurodegenerative disorders
3. The role of iron and copper molecules in the neuronal vulnerability of locus coeruleus and substantia nigra during aging
4. Effect of Familial Parkinson's Disease Point Mutations A30P and A53T on the Structural Properties, Aggregation, and Fibrillation of Human α-Synuclein
5. The A53T α-Synuclein Mutation Increases Iron-Dependent Aggregation and Toxicity
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