Antibody against Small Aggregated Peptide Specifically Recognizes Toxic Aβ-42 Oligomers in Alzheimer’s Disease-Reference-Cited by-同舟云学术

Antibody against Small Aggregated Peptide Specifically Recognizes Toxic Aβ-42 Oligomers in Alzheimer’s Disease

Published:2015-10-08 Issue:12 Volume:6 Page:1981-1989
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Bodani Riddhi U.¹,Sengupta Urmi¹,Castillo-Carranza Diana L.¹,Guerrero-Muñoz Marcos J.¹,Gerson Julia E.¹,Rudra Jai¹,Kayed Rakez¹

Affiliation:

1. Mitchell Center for Neurodegenerative Diseases, ‡Departments of Neurology and Neuroscience and Cell Biology, §Department of Pharmacology and Toxicology, and ∥Sealy Center for Vaccine Development, University of Texas Medical Branch, Galveston, Texas 77555, United States

Funder

Mitchell Center for Neurodegenerative Diseases, University of Texas Medical Branch at Galveston

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.5b00231

Reference43 articles.

1. Aβ40 and Aβ42 Amyloid Fibrils Exhibit Distinct Molecular Recycling Properties

2. The carboxy terminus of the .beta. amyloid protein is critical for the seeding of amyloid formation: Implications for the pathogenesis of Alzheimer's disease

3. Direct Observations of Amyloid β Self-Assembly in Live Cells Provide Insights into Differences in the Kinetics of Aβ(1–40) and Aβ(1–42) Aggregation