A Novel Multifunctional Compound Camellikaempferoside B Decreases Aβ Production, Interferes with Aβ Aggregation, and Prohibits Aβ-Mediated Neurotoxicity and Neuroinflammation-Reference-Cited by-同舟云学术

A Novel Multifunctional Compound Camellikaempferoside B Decreases Aβ Production, Interferes with Aβ Aggregation, and Prohibits Aβ-Mediated Neurotoxicity and Neuroinflammation

Published:2016-04-07 Issue:4 Volume:7 Page:505-518
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Yang Shigao¹,Liu Wen¹²,Lu Shuai¹,Tian Yong-zhen³,Wang Wei-yun²,Ling Tie-jun³,Liu Rui-tian¹

Affiliation:

1. National Key Laboratory of Biochemical Engineering, Institute of Process Engineering, Chinese Academy of Sciences, Beijing 100190, China

2. School of Life Science, Anhui Agricultural University, Hefei 230036, China

3. State Key Laboratory of Tea Plant Biology and Utilization, Anhui Agricultural University 130 West Changjiang Rd., Hefei 230036, P. R. China

Funder

Ministry of Science and Technology of the People's Republic of China

National Natural Science Foundation of China

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.6b00091

Reference52 articles.

1. Alzheimer's Disease: From Pathology to Therapeutic Approaches

2. The secretases: enzymes with therapeutic potential in Alzheimer disease

3. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

4. Soluble oligomers of amyloid-β peptide induce neuronal apoptosis by activating a cPLA2-dependent sphingomyelinase-ceramide pathway

5. -Amyloid-Induced Neuronal Apoptosis Involves c-Jun N-Terminal Kinase-Dependent Downregulation of Bcl-w

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