Neuroprotective Effect of a New 7,8-Dihydroxycoumarin-Based Fe2+/Cu2+ Chelator in Cell and Animal Models of Parkinson’s Disease-Reference-Cited by-同舟云学术

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Neuroprotective Effect of a New 7,8-Dihydroxycoumarin-Based Fe2+/Cu2+ Chelator in Cell and Animal Models of Parkinson’s Disease

Published:2016-11-16 Issue:1 Volume:8 Page:178-185
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Aguirre Pabla¹,García-Beltrán Olimpo²,Tapia Victoria¹,Muñoz Yorka¹,Cassels Bruce K.³,Núñez Marco T.¹

Affiliation:

1. Biology Department, Faculty of Sciences, Universidad de Chile, Santiago 7800024, Chile

2. Facultad de Ciencias Naturales y Matemáticas, Universidad de Ibagué, Ibagué 730001, Colombia

3. Department of Chemistry, Faculty of Sciences, Universidad de Chile, Santiago 7800024, Chile

Funder

Comisión Nacional de Investigación Científica y Tecnológica

Fondo Nacional de Desarrollo Científico y Tecnológico

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.6b00309

Reference45 articles.

1. Iron deposits in the chronically inflamed central nervous system and contributes to neurodegeneration

2. ALTERATIONS IN THE LEVELS OF IRON, FERRITIN AND OTHER TRACE METALS IN PARKINSON'S DISEASE AND OTHER NEURODEGENERATIVE DISEASES AFFECTING THE BASAL GANGLIA

3. Excess iron harms the brain: the syndromes of neurodegeneration with brain iron accumulation (NBIA)

4. Increased iron (III) and total iron content in post mortem substantia nigra of parkinsonian brain

5. Morphological alterations of the synapses in the locus coeruleus in Parkinson's disease

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