Gene Dysfunction Mediates Immune Response to Dopaminergic Degeneration in Parkinson’s Disease-Reference-Cited by-同舟云学术

Gene Dysfunction Mediates Immune Response to Dopaminergic Degeneration in Parkinson’s Disease

Published:2018-10-05 Issue:2 Volume:10 Page:803-811
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Jiao Zhigang¹²,Zhang Wenlong³,Chen Chaojun⁴,Zhu Xiaoqin⁵,Chen Xiang³,Zhou Miaomiao³,Peng Guoyou³,Liu Hanqun³,Qiu Jiewen³,Lin Yuwan³,Huang Shuxuan³,Mo Mingshu³,Yang Xinling⁶,Qu Shaogang¹²,Xu Pingyi³^ORCID

Affiliation:

1. Central Laboratory, Shunde Hospital, Southern Medical University, Foshan 528300, China

2. Key Laboratory of Mental Health of the Ministry of Education, Southern Medical University, Foshan 528300, China

3. Department of Neurology, the First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China

4. Department of Neurology, Guangzhou Chinese Medical Integrated Hospital (Huadu), Guangdong 510800, China

5. Guangzhou Medical University, Guangzhou 511436, China

6. Department of Neurology, the Third Affiliated Hospital of Xinjiang Medical University, Urumqi 830011, China

Funder

Ministry of Science and Technology of the People's Republic of China

Ministry of Education of the People's Republic of China

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Guangzhou City

Guangzhou Science and Technology Bureau

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.8b00373

Reference103 articles.

1. Aging and Parkinson's disease: Different sides of the same coin?

2. Microglia Polarization, Gene-Environment Interactions and Wnt/β-Catenin Signaling: Emerging Roles of Glia-Neuron and Glia-Stem/Neuroprogenitor Crosstalk for Dopaminergic Neurorestoration in Aged Parkinsonian Brain

3. Parkinson's disease

4. The potential role of neuroinflammation and transcription factors in Parkinson disease

5. Staging of brain pathology related to sporadic Parkinson’s disease

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