Azelnidipine Attenuates the Oxidative and NFκB Pathways in Amyloid-β-Stimulated Cerebral Endothelial Cells-Reference-Cited by-同舟云学术

Azelnidipine Attenuates the Oxidative and NFκB Pathways in Amyloid-β-Stimulated Cerebral Endothelial Cells

Published:2018-11-06 Issue:1 Volume:10 Page:209-215
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Teng Tao¹,Ridgley Devin M.¹,Tsoy Andrey²,Sun Grace Y.³,Askarova Sholpan²,Lee James C.¹^ORCID

Affiliation:

1. Richard and Loan Hill Department of Bioengineering, University of Illinois at Chicago, 851 South Morgan Street, MC 063, Chicago, Illinois 60607, United States

2. National Laboratory Astana, Nazarbayev University, Astana, Kazakhstan 010000

3. Department of Biochemistry, University of Missouri, Columbia, Missouri 65211, United States

Funder

National Institute on Aging

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.8b00368

Reference42 articles.

1. Amyloid Beta Induces Oxidative Stress-Mediated Blood–Brain Barrier Changes in Capillary Amyloid Angiopathy

2. Intravenous injection of beta-amyloid seeds promotes cerebral amyloid angiopathy (CAA)

3. Capillary cerebral amyloid angiopathy in Alzheimer’s disease: association with allocortical/hippocampal microinfarcts and cognitive decline

4. Mixed oligomers and monomeric amyloid-β disrupts endothelial cells integrity and reduces monomeric amyloid-β transport across hCMEC/D3 cell line as an in vitro blood–brain barrier model

5. Amyloid-Beta Disrupts Calcium and Redox Homeostasis in Brain Endothelial Cells

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