SQSTM1/p62: A Potential Target for Neurodegenerative Disease-Reference-Cited by-同舟云学术

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SQSTM1/p62: A Potential Target for Neurodegenerative Disease

Published:2019-01-18 Issue:5 Volume:10 Page:2094-2114
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Ma Shifan,Attarwala Insiya Y.,Xie Xiang-Qun¹^ORCID

Affiliation:

1. ID4Pharma LLC, Bridgeville, Pennsylvania 15017, United States

Funder

U.S. Department of Defense

National Institute on Drug Abuse

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.8b00516

Reference207 articles.

1. Trends in the Molecular Pathogenesis and Clinical Therapeutics of Common Neurodegenerative Disorders

2. Genomic structure and promoter analysis of the p62 gene encoding a non-proteasomal multiubiquitin chain binding protein

3. p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy

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2. Proximity labeling reveals dynamic changes in the SQSTM1 protein network;Journal of Biological Chemistry;2024-09

3. Genetic investigation of patients with autosomal recessive ataxia and identification of two novel variants in the SQSTM1 and SYNE1 genes;Human Genome Variation;2024-08-30

4. Genotype–phenotype correlation ofSQSTM1variants in patients with amyotrophic lateral sclerosis;Journal of Medical Genetics;2024-08-08

5. Absence of motor impairments or pathological changes in TMEM230 knockout rats;Neuroscience Letters;2024-08

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