Breaking the Iron Homeostasis: A “Trojan Horse” Self-Assembled Nanodrug Sensitizes Homologous Recombination Proficient Ovarian Cancer Cells to PARP Inhibition-Reference-Cited by-同舟云学术

Breaking the Iron Homeostasis: A “Trojan Horse” Self-Assembled Nanodrug Sensitizes Homologous Recombination Proficient Ovarian Cancer Cells to PARP Inhibition

Published:2022-08-03 Issue:8 Volume:16 Page:12786-12800
ISSN:1936-0851
Container-title:ACS Nano
language:en
Short-container-title:ACS Nano

Author:

Li Yangyang¹,Cen Yixuan¹,Fang Yifeng²,Tang Sangsang¹,Li Sen¹,Ren Yan¹,Zhang Hongbo³⁴^ORCID,Lu Weiguo¹⁵⁶,Xu Junfen¹⁵

Affiliation:

1. Women’s Reproductive Health Laboratory of Zhejiang Province, Women’s Hospital, Zhejiang University School of Medicine, Hangzhou 310006, Zhejiang, China

2. Department of General Surgery, Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, Hangzhou 310016, Zhejiang, China

3. Pharmaceutical Sciences Laboratory, Åbo Akademi University, Turku FI-20520, Finland

4. Turku Bioscience Centre, University of Turku and Åbo Akademi University, Turku FI-20520, Finland

5. Department of Gynecologic Oncology, Women’s Hospital, Zhejiang University School of Medicine, Hangzhou 310006, ZhejiangChina

6. Cancer Center, Zhejiang University, Hangzhou 310058, Zhejiang, China

Funder

Central University Basic Research Fund of China

Academy of Finland

Opetus- ja Kulttuuriministeri?

Beijing Kanghua Foundation for the Development of Traditional Chinese and Western Medicine

Publisher

American Chemical Society (ACS)

Subject

General Physics and Astronomy,General Engineering,General Materials Science

Link

https://pubs.acs.org/doi/pdf/10.1021/acsnano.2c04956

Reference47 articles.

1. Cancer statistics, 2020

2. Microenvironment remodeled by tumor and stromal cells elevates fibroblast-derived COL1A1 and facilitates ovarian cancer metastasis

3. Modular Peptide Probe for Pre/Intra/Postoperative Therapeutic to Reduce Recurrence in Ovarian Cancer

4. Maintenance Olaparib in Patients with Newly Diagnosed Advanced Ovarian Cancer

5. Homologous Recombination Deficiency: Exploiting the Fundamental Vulnerability of Ovarian Cancer

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