Nanoparticles Mediating the Sustained Puerarin Release Facilitate Improved Brain Delivery to Treat Parkinson’s Disease-Reference-Cited by-同舟云学术

Nanoparticles Mediating the Sustained Puerarin Release Facilitate Improved Brain Delivery to Treat Parkinson’s Disease

Published:2019-10-22 Issue:48 Volume:11 Page:45276-45289
ISSN:1944-8244
Container-title:ACS Applied Materials & Interfaces
language:en
Short-container-title:ACS Appl. Mater. Interfaces

Author:

Chen Tongkai¹²^ORCID,Liu Wei¹²,Xiong Sha¹²,Li Dongli¹²,Fang Shuhuan¹²,Wu Zhenfeng³,Wang Qi¹²,Chen Xiaojia⁴

Affiliation:

1. Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou 510405, China

2. Institute of Clinical Pharmacology, Guangzhou University of Chinese Medicine, Guangzhou 510405, China

3. Key Laboratory of Modern Preparation of Traditional Chinese Medicine, Ministry of Education, Jiangxi University of Traditional Chinese Medicine, Nanchang 330004, China

4. State Key Laboratory of Quality Research in Chinese Medicine, Institute of Chinese Medical Sciences, University of Macau, Macau 999078, China

Funder

Universidade de Macau

Guangzhou Science, Technology and Innovation Commission

Natural Science Foundation of Guangdong Province

National Natural Science Foundation of China

Publisher

American Chemical Society (ACS)

Subject

General Materials Science

Link

https://pubs.acs.org/doi/pdf/10.1021/acsami.9b16047

Reference48 articles.

1. Midbrain Gene Screening Identifies a New Mesoaccumbal Glutamatergic Pathway and a Marker for Dopamine Cells Neuroprotected in Parkinson’s Disease

2. Neuroprotection by Paeoniflorin in the MPTP mouse model of Parkinson's disease

3. Neuroprotective properties of icariin in MPTP-induced mouse model of Parkinson's disease: Involvement of PI3K/Akt and MEK/ERK signaling pathways

4. Modulatory effects of resveratrol on endoplasmic reticulum stress-associated apoptosis and oxido-inflammatory markers in a rat model of rotenone-induced Parkinson's disease

5. Puerarin attenuates neuronal degeneration in the substantia nigra of 6-OHDA-lesioned rats through regulating BDNF expression and activating the Nrf2/ARE signaling pathway

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