β-Cyclodextrin-poly (β-Amino Ester) Nanoparticles Are a Generalizable Strategy for High Loading and Sustained Release of HDAC Inhibitors-Reference-Cited by-同舟云学术

β-Cyclodextrin-poly (β-Amino Ester) Nanoparticles Are a Generalizable Strategy for High Loading and Sustained Release of HDAC Inhibitors

Published:2021-04-27 Issue:18 Volume:13 Page:20960-20973
ISSN:1944-8244
Container-title:ACS Applied Materials & Interfaces
language:en
Short-container-title:ACS Appl. Mater. Interfaces

Author:

Chaudhuri Sauradip¹^ORCID,Fowler Martha J.¹,Baker Cassandra¹,Stopka Sylwia A.²³,Regan Michael S.²,Sablatura Lindsey¹,Broughton Colton W.¹,Knight Brandon E.¹,Stabenfeldt Sarah E.⁴,Agar Nathalie Y. R.²³⁵^ORCID,Sirianni Rachael W.¹⁴

Affiliation:

1. Vivian L. Smith Department of Neurosurgery, University of Texas Health Science Center at Houston, Houston, Texas 77030, United States

2. Department of Neurosurgery, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts 02115, United States

3. Department of Radiology, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts 02115, United States

4. School of Biological and Health Systems Engineering, Arizona State University, Tempe, Arizona 85281, United States

5. Department of Cancer Biology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts 02215, United States

Funder

National Cancer Institute

National Institute of Neurological Disorders and Stroke

National Institute of Biomedical Imaging and Bioengineering

Pediatric Brain Tumor Foundation

Eunice Kennedy Shriver National Institute of Child Health and Human Development

Ians Friends Foundation

Publisher

American Chemical Society (ACS)

Subject

General Materials Science

Link

https://pubs.acs.org/doi/pdf/10.1021/acsami.0c22587

Reference46 articles.

1. Expression of nuclear receptor corepressors and class I histone deacetylases in astrocytic gliomas

2. Inhibition of histone deacetylases in cancer therapy: lessons from leukaemia

3. Histone deacetylase inhibitors for the treatment of cancer stem cells

4. Inhibition of Histone Deacetylase Impacts Cancer Stem Cells and Induces Epithelial-Mesenchyme Transition of Head and Neck Cancer

5. Histone deacetylase inhibitors promote glioma cell death by G2 checkpoint abrogation leading to mitotic catastrophe

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