Proteomics Identified Overexpression of SET Oncogene Product and Possible Therapeutic Utility of Protein Phosphatase 2A in Alveolar Soft Part Sarcoma
Author:
Affiliation:
1. Division of Pharmacoproteomics, National Cancer Center Research Institute, Tsukiji 5-1-1, Chuo-ku, Tokyo 104-0045, Japan
Publisher
American Chemical Society (ACS)
Subject
General Chemistry,Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/pr400929h
Reference37 articles.
1. Alveolar soft-part sarcomas.Structurally characteristic tumors of uncertain histogenesis
2. The der(17)t(X;17)(p11;q25) of human alveolar soft part sarcoma fuses the TFE3 transcription factor gene to ASPL, a novel gene at 17q25
3. Angiogenesis-Promoting Gene Patterns in Alveolar Soft Part Sarcoma
4. Gene expression profiling of alveolar soft-part sarcoma (ASPS)
5. Bioinformatic Analysis of Patient-Derived ASPS Gene Expressions and ASPL-TFE3 Fusion Transcript Levels Identify Potential Therapeutic Targets
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1. SET Protein in Cancer: A Potential Therapeutic Target;Mini-Reviews in Medicinal Chemistry;2021-11-23
2. Sphingosine Analogs and Protein Phosphatase 2A as a Molecular Targeted Cancer Therapy: A Mini Systematic Review;Clinical Cancer Drugs;2020-11-06
3. Proteomic research in sarcomas – current status and future opportunities;Seminars in Cancer Biology;2020-04
4. Systematic screening identifies a 2‐gene signature as a high‐potential prognostic marker of undifferentiated pleomorphic sarcoma/myxofibrosarcoma;Journal of Cellular and Molecular Medicine;2019-11-19
5. Protein phosphatase 2A (PP2A): a key phosphatase in the progression of chronic obstructive pulmonary disease (COPD) to lung cancer;Respiratory Research;2019-10-17
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