Molecular Mechanism Behind the Resistance of the G1202R-Mutated Anaplastic Lymphoma Kinase to the Approved Drug Ceritinib
Author:
Affiliation:
1. Department of Thoracic Cardiovascular, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China
Funder
Natural Science Foundation of Zhejiang Province
Wenzhou Public Welfare Science and Technology Plan
Publisher
American Chemical Society (ACS)
Subject
Materials Chemistry,Surfaces, Coatings and Films,Physical and Theoretical Chemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/acs.jpcb.8b02040
Reference56 articles.
1. EGFR Mutations in Lung Cancer: Correlation with Clinical Response to Gefitinib Therapy
2. Acquired Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib Is Associated with a Second Mutation in the EGFR Kinase Domain
3. Anaplastic Lymphoma Kinase Inhibition in Non–Small-Cell Lung Cancer
4. Activity of Crizotinib (PF02341066), a Dual Mesenchymal-Epithelial Transition (MET) and Anaplastic Lymphoma Kinase (ALK) Inhibitor, in a Non-small Cell Lung Cancer Patient with De Novo MET Amplification
5. Crizotinib versus Chemotherapy in AdvancedALK-Positive Lung Cancer
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