Drosophila Frataxin: An Iron Chaperone during Cellular Fe−S Cluster Bioassembly
Author:
Affiliation:
1. Department of Biochemistry and Molecular Biology, Wayne State University, School of Medicine, Detroit, Michigan 48201, and Department of Medicine, Division of Hematology-Oncology, University of Pennsylvania, Philadelphia, Pennsylvania 19104
Publisher
American Chemical Society (ACS)
Subject
Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/bi800366d
Reference57 articles.
1. Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion
2. Regulation of Mitochondrial Iron Accumulation by Yfh1p, a Putative Homolog of Frataxin
3. Inactivation of the Friedreich ataxia mouse gene leads to early embryonic lethality without iron accumulation
4. The yeast frataxin homolog Yfh1p plays a specific role in the maturation of cellular Fe/S proteins
5. Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxia
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