Nuclear Leukocyte Immunoglobulin-like Receptor A3 Is Monomeric and Is Involved in Multiple Layers of Regulated Gene Expression and Translation-Reference-Cited by-同舟云学术

Nuclear Leukocyte Immunoglobulin-like Receptor A3 Is Monomeric and Is Involved in Multiple Layers of Regulated Gene Expression and Translation

Published:2021-04-01 Issue:6 Volume:20 Page:3078-3089
ISSN:1535-3893
Container-title:Journal of Proteome Research
language:en
Short-container-title:J. Proteome Res.

Author:

An Hongyan¹,Richardson Alexander¹,Rajasekariah Poornima¹,Zhong Ling²,Fernando Bentotage Samitha M.¹,Macmillan Alexander³,Klotzsch Enrico⁴⁵^ORCID,Bryant Katherine¹,Kaakoush Nadeem O.¹,Tedla Nicodemus¹^ORCID

Affiliation:

1. Mechanisms of Diseases and Translational Research, School of Medical Sciences, Department of Pathology, UNSW, Sydney 2052, Australia

2. Bioanalytical Mass Spectrometry Facility, Mark Wainwright Analytical Centre, UNSW, Sydney 2052, Australia

3. Biomedical Imaging Facility, Mark Wainwright Analytical Centre, UNSW, Sydney 2052, Australia

4. Laboratory of Applied Mechanobiology, Department for Health Sciences and Technology, ETH Zürich, Zürich 8093, Switzerland

5. Institute for Biology, Experimental Biophysics/Mechanobiology, Humboldt University of Berlin, Berlin 10115, German

Funder

National Health and Medical Research Council

Publisher

American Chemical Society (ACS)

Subject

General Chemistry,Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.jproteome.0c00946

Reference33 articles.

1. The LILR family: modulators of innate and adaptive immune pathways in health and disease

2. Serum Leukocyte Immunoglobulin-Like Receptor A3 (LILRA3) Is Increased in Patients with Multiple Sclerosis and Is a Strong Independent Indicator of Disease Severity; 6.7kbp LILRA3 Gene Deletion Is Not Associated with Diseases Susceptibility

3. Soluble LILRA3, a Potential Natural Antiinflammatory Protein, Is Increased in Patients with Rheumatoid Arthritis and Is Tightly Regulated by Interleukin 10, Tumor Necrosis Factor-α, and Interferon-γ

4. Glycosylation in a Mammalian Expression System Is Critical for the Production of Functionally Active Leukocyte Immunoglobulin-like Receptor A3 Protein

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