Familial Hypertrophic Cardiomyopathy Can Be Characterized by a Specific Pattern of Orientation Fluctuations of Actin Molecules,-Reference-Cited by-同舟云学术

Familial Hypertrophic Cardiomyopathy Can Be Characterized by a Specific Pattern of Orientation Fluctuations of Actin Molecules,

Published:2010-06-04 Issue:25 Volume:49 Page:5269-5277
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Borejdo J.¹,Szczesna-Cordary D.²,Muthu P.²,Calander N.³

Affiliation:

1. Department of Molecular Biology and Immunology and Center for Commercialization of Fluorescence Technologies, University of North Texas Health Science Center, 3500 Camp Bowie Boulevard, Fort Worth, Texas 76107

2. Department of Molecular and Cellular Pharmacology, University of Miami, Miller School of Medicine, 1600 NW 10th Avenue, Miami, Florida 33136

3. Department of Physics, Macquarie University, Sydney, NSW 2109, Australia

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi1006749

Reference39 articles.

1. Mutations in either the essential or regulatory light chains of myosin are associated with a rare myopathy in human heart and skeletal muscle

2. Identification of two novel mutations in the ventricular regulatory myosin light chain gene ( MYL2 ) associated with familial and classical forms of hypertrophic cardiomyopathy

3. Myosin light chain mutations in familial hypertrophic cardiomyopathy: phenotypic presentation and frequency in Danish and South African populations

4. Systematic analysis of the regulatory and essential myosin light chain genes: genetic variants and mutations in hypertrophic cardiomyopathy

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