Hypertriglyceridemic VLDL Decreases Plasminogen Binding to Endothelial Cells and Surface-Localized Fibrinolysis-Reference-Cited by-同舟云学术

Hypertriglyceridemic VLDL Decreases Plasminogen Binding to Endothelial Cells and Surface-Localized Fibrinolysis

Published:1996-01-01 Issue:19 Volume:35 Page:6080-6088
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Li Xin-Nong¹,Koons Jay C.¹,Benza Raymond L.¹,Parks J. Michael¹,Varma Vivek K.¹,Bradley William A.¹,Gianturco Sandra H.¹,Taylor Kenneth B.¹,Grammer J. Robert¹,Tabengwa Edlue M.¹,Booyse Francois M.¹

Affiliation:

1. Department of Medicine, Divisions of Cardiovascular Disease and Geriatric Medicine/Gerontology and Department of Biochemistry, University of Alabama at Birmingham, Birmingham, Alabama 35294-2170

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi952032i

Reference60 articles.

1. Characterization of human endothelial cell urokinase-type plasminogen activator receptor protein and messenger RNA

2. Characterization of tissue plasminogen activator binding proteins isolated from endothelial cells and other cell types

3. Lipoprotein(a): relation to other risk factors and genetic heritability. Results from a Dutch parent-twin study

4. Cultured aortic endothelial cells from pigs with von Willebrand disease: in vitro model for studying the molecular defect(s) of the disease.

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