The 60 kDa Insulin Receptor Substrate Functions Like an IRS Protein (pp60IRS3) in Adipose Cells-Reference-Cited by-同舟云学术

The 60 kDa Insulin Receptor Substrate Functions Like an IRS Protein (pp60IRS3) in Adipose Cells

Published:1997-07-01 Issue:27 Volume:36 Page:8304-8310
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Smith-Hall Jennifer¹,Pons Sebastian¹,Patti Mary Elizabeth¹,Burks Deborah J.¹,Yenush Lynne¹,Sun Xiao Jian¹,Kahn C. Ronald¹,White Morris F.¹

Affiliation:

1. Joslin Diabetes Center and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02215, and Department of Physiology and Biophysics, Indiana University School of Medicine, Indianapolis, Indiana 46202

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi9630974

Reference46 articles.

1. Alternative pathway of insulin signalling in mice with targeted disruption of the IRS-1 gene

2. Phosphatidylinositol 3′-kinase is activated by association with IRS-1 during insulin stimulation.

3. Association of IRS-1 with the insulin receptor and the phosphatidylinositol 3'-kinase. Formation of binary and ternary signaling complexes in intact cells.

4. Insulin stimulates tyrosine phosphorylation of its receptor β-subunit in intact rat hepatocytes

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