Preferentially Increased Nitration of α-Synuclein at Tyrosine-39 in a Cellular Oxidative Model of Parkinson’s Disease
Author:
Affiliation:
1. Buck Institute for Age Research, 8001 Redwood Boulevard, Novato, California 94945
Publisher
American Chemical Society (ACS)
Subject
Analytical Chemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/ac901176t
Reference50 articles.
1. Mutation in the α-Synuclein Gene Identified in Families with Parkinson's Disease
2. Protein Nitration in Parkinsonʼs Disease
3. Oxidative Damage Linked to Neurodegeneration by Selective α-Synuclein Nitration in Synucleinopathy Lesions
4. Oxidative post-translational modifications of α-synuclein in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) mouse model of Parkinson's disease
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