Comparative Transcriptome and Proteome Analysis of Ha-ras and B-raf Mutated Mouse Liver Tumors-Reference-Cited by-同舟云学术

Comparative Transcriptome and Proteome Analysis of Ha-ras and B-raf Mutated Mouse Liver Tumors

Published:2009-06-17 Issue:8 Volume:8 Page:3987-3994
ISSN:1535-3893
Container-title:Journal of Proteome Research
language:en
Short-container-title:J. Proteome Res.

Author:

Rignall Benjamin¹,Ittrich Carina¹,Krause Eberhard¹,Appel Klaus E.¹,Buchmann Albrecht¹,Schwarz Michael¹

Affiliation:

1. Department of Toxicology, Institute of Experimental and Clinical Pharmacology and Toxicology, University of Tübingen, Wilhelmstr. 56, 72074 Tübingen, Germany, German Cancer Research Center, Central Unit of Biostatistics, Im Neuenheimer Feld 280, 69120 Heidelberg, Germany, Leibniz Institute for Molecular Pharmacology (FMP), Robert-Rössle-Strasse 10, 13125 Berlin, Germany, and Federal Institute for Risk Assessment, Center for Experimental Toxicology, Thielallee 88-92, 14195 Berlin, Germany

Publisher

American Chemical Society (ACS)

Subject

General Chemistry,Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/pr9002933

Reference36 articles.

1. Selective pressure during tumor promotion by phenobarbital leads to clonal outgrowth of β-catenin-mutated mouse liver tumors

2. Activation of β-catenin provides proliferative and invasive advantages in c-myc /TGF-α hepatocarcinogenesis promoted by phenobarbital

3. PCB 153, a Non-dioxin–like Tumor Promoter, Selects for β-Catenin (Catnb)–Mutated Mouse Liver Tumors

4. Mutational activation of the c-Ha-ras gene in liver tumors of different rodent strains: correlation with susceptibility to hepatocarcinogenesis.

5. B-Raf and Ha-ras mutations in chemically induced mouse liver tumors

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