β-Elemene Attenuates Fibrosis after Esophageal Endoscopic Submucosal Dissection via Modulating the HIF-1α/HK2/p38-MAPK Signaling Axis
Author:
Affiliation:
1. Department of Gastroenterology, The Second Affiliated Hospital of Fujian Medical University, Quanzhou 362000, China
Funder
Natural Science Foundation of Fujian Province
Quanzhou City, Fujian Province
Publisher
American Chemical Society (ACS)
Subject
Biomedical Engineering,Biomaterials
Link
https://pubs.acs.org/doi/pdf/10.1021/acsbiomaterials.1c00047
Reference25 articles.
1. Endoscopic resection is the criterion standard of treatment for patients with early squamous cell neoplasia of the esophagus
2. Risk factors for postoperative stricture after endoscopic submucosal dissection for superficial esophageal carcinoma
3. Preventing Stricture Formation by Covered Esophageal Stent Placement After Endoscopic Submucosal Dissection for Early Esophageal Cancer
4. Independent risk factors for esophageal refractory stricture after extensive endoscopic submucosal dissection
5. Esophageal Stricture: Incidence, Treatment Patterns, and Recurrence Rate
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1. β-elemene alleviates esophageal fibrosis after endoscopic submucosal dissection via the FAP-mediated PTEN-PI3K/AKT signaling pathway;Heliyon;2024-05
2. PDLIM1 interacts with HK2 to promote gastric cancer progression through enhancing the Warburg effect via Wnt/β-catenin signaling;Cell and Tissue Research;2023-11-06
3. Combined transcriptome and proteome analysis of yak PASMCs under hypoxic and normoxic conditions;PeerJ;2022-11-25
4. β-Elemene Attenuates Renal Fibrosis in the Unilateral Ureteral Obstruction Model by Inhibition of STAT3 and Smad3 Signaling via Suppressing MyD88 Expression;International Journal of Molecular Sciences;2022-05-16
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