Ca2+-Dependent Interaction with Calmodulin Is Conserved in the Synapsin Family: Identification of a High-Affinity Site
Author:
Affiliation:
1. Research School of Biosciences, University of Kent, Canterbury, Kent, CT2 7NJ, England
Publisher
American Chemical Society (ACS)
Subject
Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/bi970709r
Reference41 articles.
1. Synapsin I deficiency results in the structural change in the presynaptic terminals in the murine nervous system.
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