Mechanism of Cardiac Tropomyosin Transitions on Filamentous Actin As Revealed by All-Atom Steered Molecular Dynamics Simulations-Reference-Cited by-同舟云学术

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Mechanism of Cardiac Tropomyosin Transitions on Filamentous Actin As Revealed by All-Atom Steered Molecular Dynamics Simulations

Published:2018-06-04 Issue:12 Volume:9 Page:3301-3306
ISSN:1948-7185
Container-title:The Journal of Physical Chemistry Letters
language:en
Short-container-title:J. Phys. Chem. Lett.

Author:

Williams Michael R.¹,Tardiff Jil C.²³,Schwartz Steven D.¹^ORCID

Affiliation:

1. Department of Chemistry and Biochemistry, The University of Arizona, Tucson, Arizona 85721, United States

2. Department of Medicine, The University of Arizona, Tucson, Arizona 85724, United States

3. Department of Cellular and Molecular Medicine, The University of Arizona, Tucson, Arizona 85724, United States

Funder

National Heart, Lung, and Blood Institute

Publisher

American Chemical Society (ACS)

Subject

General Materials Science,Physical and Theoretical Chemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.jpclett.8b00958

Reference25 articles.

1. Atomic resolution probe for allostery in the regulatory thin filament

2. Actin in a twist

3. 50 years of fiber diffraction

4. The Shape and Flexibility of Tropomyosin Coiled Coils: Implications for Actin Filament Assembly and Regulation

5. Regulation of the interaction between actin and myosin subfragment 1: evidence for three states of the thin filament

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2. Modeling Human Cardiac Thin Filament Structures;Frontiers in Physiology;2022-06-22

3. Structure and Dynamics of the Flexible Cardiac Troponin T Linker Domain in a Fully Reconstituted Thin Filament;Biochemistry;2022-06-13

4. Computational and biophysical determination of pathogenicity of variants of unknown significance in cardiac thin filament;JCI Insight;2021-12-08

5. Loss of crossbridge inhibition drives pathological cardiac hypertrophy in patients harboring the TPM1 E192K mutation;Journal of General Physiology;2021-07-28

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