DNA Polymerase II Supports the Replicative Bypass of N2-Alkyl-2′-deoxyguanosine Lesions in Escherichia coli Cells
Author:
Funder
National Institute of Environmental Health Sciences
Publisher
American Chemical Society (ACS)
Subject
Toxicology,General Medicine
Link
https://pubs.acs.org/doi/pdf/10.1021/acs.chemrestox.0c00478
Reference25 articles.
1. Chemical biology of mutagenesis and DNA repair: cellular responses to DNA alkylation
2. Balancing repair and tolerance of DNA damage caused by alkylating agents
3. Reactions of Formaldehyde Plus Acetaldehyde with Deoxyguanosine and DNA: Formation of Cyclic Deoxyguanosine Adducts and Formaldehyde Cross-Links
4. DNA adducts from acetaldehyde: implications for alcohol-related carcinogenesis
5. Identification of DNA Adducts of Methylglyoxal
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2. 8-OxodGuo and Fapy•dG Mutagenicity in Escherichia coli Increases Significantly when They Are Part of a Tandem Lesion with 5-Formyl-2′-deoxyuridine;Chemical Research in Toxicology;2024-07-23
3. Escherichia coli DNA replication: the old model organism still holds many surprises;FEMS Microbiology Reviews;2024-06-20
4. Mass spectrometry-based assays for assessing replicative bypass and repair of DNA alkylation in cells;RSC Advances;2023
5. Correction to DNA Polymerase II Supports the Replicative Bypass of N2-Alkyl-2′-deoxyguanosine Lesions in Escherichia coli Cells;Chemical Research in Toxicology;2021-09-21
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