Long-Term Triclocarban Exposure Induced Enterotoxicity by Triggering Intestinal AhR-Mediated Inflammation and Disrupting Microbial Community in Mice-Reference-Cited by-同舟云学术

Long-Term Triclocarban Exposure Induced Enterotoxicity by Triggering Intestinal AhR-Mediated Inflammation and Disrupting Microbial Community in Mice

Published:2024-03-25 Issue:4 Volume:37 Page:658-668
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Song Yuchen¹²,Lei Hehua¹²,Cao Zheng¹²,Zhang Cui¹²,Chen Chuan¹²,Wu Mengjing¹³,Zhang Huabao¹,Du Ruichen¹²,Lijun Liu¹²,Chen Xiaoyu³,Zhang Limin¹²^ORCID

Affiliation:

1. State Key Laboratory of Magnetic Resonance and Imaging, National Centre for Magnetic Resonance in Wuhan, Innovation Academy of Precision Measurement Science and Technology, Chinese Academy of Sciences (CAS), Wuhan 430071, China

2. University of Chinese Academy of Sciences, Beijing 100049, China

3. The People’s Hospital of Guangxi Zhuang Autonomous Region & Guangxi Academy of Medical Sciences, Nanning 530021, Guangxi, China

Funder

Foshan Core Technology Tackling Key Project

Strategic Priority Research Program

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.chemrestox.4c00042

Reference42 articles.

1. Triclocarban shifted the microbial communities and promoted the spread of antibiotic resistance genes in nitrifying granular sludge system

2. Fate of Triclocarban (TCC) in aquatic and terrestrial systems and human exposure

3. Fate, risk and removal of triclocarban: A critical review

4. Triclocarban and triclosan exacerbate high-fat diet-induced hepatic lipid accumulation at environmental related levels: The potential roles of estrogen-related receptors pathways

5. Triclocarban triggers osteoarthritis via DNMT1-mediated epigenetic modification and suppression of COL2A in cartilage tissues

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