Dynamic Phosphorylation of Apoptosis Signal Regulating Kinase 1 (ASK1) in Response to Oxidative and Electrophilic Stress-Reference-Cited by-同舟云学术

Dynamic Phosphorylation of Apoptosis Signal Regulating Kinase 1 (ASK1) in Response to Oxidative and Electrophilic Stress

Published:2016-11-30 Issue:12 Volume:29 Page:2175-2183
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Morales Betanzos Carlos¹,Federspiel Joel D.¹,Palubinsky Amy M.¹,McLaughlin BethAnn¹,Liebler Daniel C.¹^ORCID

Affiliation:

1. Department of Biochemistry, Vanderbilt University School of Medicine, ‡Neuroscience Graduate Program, Vanderbilt Brain Institute, Vanderbilt University, §Department of Neurology, Vanderbilt University, Nashville, Tennessee 37232, United States

Funder

National Institute of Environmental Health Sciences

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.chemrestox.6b00339

Reference50 articles.

1. Induction of Apoptosis by ASK1, a Mammalian MAPKKK That Activates SAPK/JNK and p38 Signaling Pathways

2. Activation mechanisms of ASK1 in response to various stresses and its significance in intracellular signaling

3. Apoptosis Signal-Regulating Kinase 1 in Stress and Immune Response

4. Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1

5. ROS-dependent activation of the TRAF6-ASK1-p38 pathway is selectively required for TLR4-mediated innate immunity

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