Insulin Signaling Disruption and INF-γ Upregulation Induce Aβ<sub>1–42</sub> and Hyperphosphorylated-Tau Proteins Synthesis and Cell Death after Paraquat Treatment of Primary Hippocampal Cells-Reference-Cited by-同舟云学术

Insulin Signaling Disruption and INF-γ Upregulation Induce Aβ_1–42 and Hyperphosphorylated-Tau Proteins Synthesis and Cell Death after Paraquat Treatment of Primary Hippocampal Cells

Published:2022-11-17 Issue:12 Volume:35 Page:2214-2218
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Abascal Maria Luisa¹,Sanjuan Javier²,Moyano Paula²,Sola Emma²,Flores Andrea²,Garcia José Manuel²,Garcia Jimena²,Frejo María Teresa²,del Pino Javier²^ORCID

Affiliation:

1. Department of Pathology, Gregorio Marañon Hospital, 28007 Madrid, Spain

2. Department of Pharmacology and Toxicology, Veterinary School, Complutense University of Madrid, 28040 Madrid, Spain

Funder

Universidad Complutense de Madrid

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.chemrestox.2c00278

Reference30 articles.

1. The toxic influence of paraquat on hippocampus of mice: Involvement of oxidative stress

2. Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2O2

3. Pathological changes in hippocampal neuronal circuits underlie age-associated neurodegeneration and memory loss: Positive clue toward SAD

4. Toxicokinetics and toxicodynamics of paraquat accumulation in mouse brain

5. Primary hippocampal neuronal cell death induction after acute and repeated paraquat exposures mediated by AChE variants alteration and cholinergic and glutamatergic transmission disruption

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