Polybrominated Diphenyl Ether Quinone Exposure Induces Atherosclerosis Progression via CD36-Mediated Lipid Accumulation, NLRP3 Inflammasome Activation, and Pyroptosis-Reference-Cited by-同舟云学术

Polybrominated Diphenyl Ether Quinone Exposure Induces Atherosclerosis Progression via CD36-Mediated Lipid Accumulation, NLRP3 Inflammasome Activation, and Pyroptosis

Published:2021-08-24 Issue:9 Volume:34 Page:2125-2134
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Wang Yuting¹,Fang Changyu¹,Xu Lei¹,Yang Bingwei¹,Song Erqun¹^ORCID,Song Yang¹²^ORCID

Affiliation:

1. Key Laboratory of Luminescence Analysis and Molecular Sensing, Ministry of Education, College of Pharmaceutical Sciences, Southwest University, 2 Tiansheng Road, Beibei District, Chongqing 400715, China

2. State Key Laboratory of Environmental Chemistry and Ecotoxicology, Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences, 18 Shuangqing Road, Haidian District, Beijing 100085, China

Funder

National Natural Science Foundation of China

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.chemrestox.1c00214

Reference56 articles.

1. Accumulation and time trends (2003–2015) of persistent organic pollutants (POPs) in blubber of finless porpoises (Neophocaena asiaeorientalis) from Korean coastal waters

2. Epigenetic Effects of Polybrominated Diphenyl Ethers on Human Health

3. Polybrominated Diphenyl Ethers in Leaves and Soil from Typical Electronic Waste Polluted Area in South China