Inhibition by Tetrahydroquinoline Sulfonamide Derivatives of the Activity of Human 8-Oxoguanine DNA Glycosylase (OGG1) for Several Products of Oxidatively induced DNA Base Lesions-Reference-Cited by-同舟云学术

Inhibition by Tetrahydroquinoline Sulfonamide Derivatives of the Activity of Human 8-Oxoguanine DNA Glycosylase (OGG1) for Several Products of Oxidatively induced DNA Base Lesions

Published:2020-12-17 Issue:1 Volume:16 Page:45-51
ISSN:1554-8929
Container-title:ACS Chemical Biology
language:en
Short-container-title:ACS Chem. Biol.

Author:

Kant Melis¹,Tahara Yu-ki²,Jaruga Pawel¹,Coskun Erdem¹³,Lloyd R. Stephen⁴,Kool Eric T.²^ORCID,Dizdaroglu Miral¹^ORCID

Affiliation:

1. Biomolecular Measurement Division, National Institute of Standards and Technology, Gaithersburg, Maryland 20899, United States

2. Department of Chemistry, ChEM-H Institute and Stanford Cancer Institute, Stanford University, Stanford, California 94305, United States

3. Institute for Bioscience and Biotechnology Research, University of Maryland, Rockville, Maryland 20850, United States

4. Oregon Institute of Occupational Health Sciences, Oregon Health and Science University, Portland, Oregon 97239, United States

Funder

National Cancer Institute

Division of Consumer and Business Services, State of Oregon

Publisher

American Chemical Society (ACS)

Subject

Molecular Medicine,General Medicine,Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acschembio.0c00877

Reference54 articles.

1. Mechanisms of free radical-induced damage to DNA

2. 5′,8-Cyclopurine Lesions in DNA Damage: Chemical, Analytical, Biological, and Diagnostic Significance

3. In vitro repair of oxidative DNA damage by human nucleotide excision repair system: Possible explanation for neurodegeneration in Xeroderma pigmentosum patients

4. Alternative nucleotide incision repair pathway for oxidative DNA damage

5. Base and Nucleotide Excision Repair of Oxidatively Generated Guanine Lesions in DNA

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