Bisaryloxime Ethers as Potent Inhibitors of Transthyretin Amyloid Fibril Formation-Reference-Cited by-同舟云学术

Bisaryloxime Ethers as Potent Inhibitors of Transthyretin Amyloid Fibril Formation

Published:2005-02-08 Issue:5 Volume:48 Page:1576-1587
ISSN:0022-2623
Container-title:Journal of Medicinal Chemistry
language:en
Short-container-title:J. Med. Chem.

Author:

Johnson Steven M.¹,Petrassi H. Michael¹,Palaninathan Satheesh K.¹,Mohamedmohaideen Nilofar N.¹,Purkey Hans E.¹,Nichols Christopher¹,Chiang Kyle P.¹,Walkup Traci¹,Sacchettini James C.¹,Sharpless K. Barry¹,Kelly Jeffery W.¹

Affiliation:

1. Department of Chemistry and the Skaggs Institute for Chemical Biology, The Scripps Research Institute, 10550 N. Torrey Pines Rd., La Jolla, Califonia 92037, and Department of Biochemistry and Biophysics, Texas A&M University, College Station, Texas 77843-2128

Publisher

American Chemical Society (ACS)

Subject

Drug Discovery,Molecular Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/jm049274d

Reference75 articles.

1. Energetic Characteristics of the New Transthyretin Variant A25T May Explain Its Atypical Central Nervous System Pathology

2. D18G Transthyretin Is Monomeric, Aggregation Prone, and Not Detectable in Plasma and Cerebrospinal Fluid: A Prescription for Central Nervous System Amyloidosis?

3. Familial meningocerebrovascular amyloidosis, Hungarian type, with mutant transthyretin (TTR Asp18Gly)

4. Familial transthyretin-type amyloid polyneuropathy in Japan: Clinical and genetic heterogeneity

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