Growth Factor Receptor-Bound Protein 14 Undergoes Light-Dependent Intracellular Translocation in Rod Photoreceptors: Functional Role in Retinal Insulin Receptor Activation-Reference-Cited by-同舟云学术

Growth Factor Receptor-Bound Protein 14 Undergoes Light-Dependent Intracellular Translocation in Rod Photoreceptors: Functional Role in Retinal Insulin Receptor Activation

Published:2009-05-29 Issue:24 Volume:48 Page:5563-5572
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Rajala Ammaji¹²,Daly Roger J.³,Tanito Masaki¹²,Allen Dustin T.¹²,Holt Lowenna J.³,Lobanova Ekaterina S.⁴,Arshavsky Vadim Y.⁴,Rajala Raju V. S.¹⁵²

Affiliation:

1. Departments of Ophthalmology

2. Dean A. McGee Eye Institute, Oklahoma City, Oklahoma 73104

3. Cancer Research Program, Garvan Institute of Medical Research, Sydney, New South Wales 2010, Australia

4. Department of Ophthalmology and Neurobiology, Duke University Medical Center, Durham, North Carolina 27710

5. Cell Biology

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi9000062

Reference41 articles.

1. Insulin Rescues Retinal Neurons from Apoptosis by a Phosphatidylinositol 3-Kinase/Akt-mediated Mechanism That Reduces the Activation of Caspase-3

2. Survival Factor-Mediated BAD Phosphorylation Raises the Mitochondrial Threshold for Apoptosis

3. Insulin-Like Growth Factor and Potassium Depolarization Maintain Neuronal Survival by Distinct Pathways: Possible Involvement of PI 3-Kinase in IGF-1 Signaling

4. Nerve Growth Factor Protects PC12 Cells Against Peroxynitrite-Induced Apoptosis via a Mechanism Dependent on Phosphatidylinositol 3-Kinase

5. In Vivo Regulation of Phosphoinositide 3-Kinase in Retina through Light-induced Tyrosine Phosphorylation of the Insulin Receptor β-Subunit

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