Proteomic Analysis of the Functional Inward Rectifier Potassium Channel (Kir) 2.1 Reveals Several Novel Phosphorylation Sites-Reference-Cited by-同舟云学术

Proteomic Analysis of the Functional Inward Rectifier Potassium Channel (Kir) 2.1 Reveals Several Novel Phosphorylation Sites

Published:2021-10-22 Issue:44 Volume:60 Page:3292-3301
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Brown Kyle A.¹²^ORCID,Anderson Corey³,Reilly Louise³,Sondhi Kunal³,Ge Ying⁴⁵^ORCID,Eckhardt Lee L.³

Affiliation:

1. Department of Surgery, University of Wisconsin-Madison, Madison, Wisconsin 53706, United States

2. Department of Chemistry, University of Wisconsin-Madison, Madison, Wisconsin 53706, United States

3. Cellular and Molecular Arrhythmia Research Program, Division of Cardiovascular Medicine, Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin 53706, United States

4. Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, Wisconsin 53705, United States

5. Human Proteomics Program, School of Medicine and Public Health, University of Wisconsin-Madison, Madison, Wisconsin 53705, United States

Funder

National Heart, Lung, and Blood Institute

Office of the Director

National Institute of General Medical Sciences

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.1c00555

Reference72 articles.

1. HRS/EHRA/APHRS Expert Consensus Statement on the Diagnosis and Management of Patients with Inherited Primary Arrhythmia Syndromes

2. What Causes Sudden Death in Heart Failure?

3. Inwardly Rectifying Potassium Channels: Their Structure, Function, and Physiological Roles

4. Cardiac potassium inward rectifier Kir2: Review of structure, regulation, pharmacology, and arrhythmogenesis

5. KCNJ2 mutation causes an adrenergic-dependent rectification abnormality with calcium sensitivity and ventricular arrhythmia

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