Methylated Cytosine Maintains G-Quadruplex Structures during Polymerase Chain Reaction and Contributes to Allelic Dropout-Reference-Cited by-同舟云学术

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Methylated Cytosine Maintains G-Quadruplex Structures during Polymerase Chain Reaction and Contributes to Allelic Dropout

Published:2017-07-10 Issue:29 Volume:56 Page:3691-3698
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Stevens Aaron J.¹^ORCID,Kennedy Martin A.¹

Affiliation:

1. Department of Pathology, University of Otago, Christchurch, New Zealand

Funder

Royal Society of New Zealand

Marsden Fund

Carney Centre for Pharmacogenomics, University of Otago

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.7b00480

Reference44 articles.

1. Allele dropout caused by a non-primer-site SNV affecting PCR amplification — A call for next-generation primer design algorithm

2. Identification and Prevention of Genotyping Errors Caused by G-Quadruplex– and i-Motif–Like Sequences

3. Inhibition of DNA synthesis by K+-stabilised G-quadruplex promotes allelic preferential amplification

4. Allele Drop-Out in the MECP2 Gene Due to G-Quadruplex and i-Motif Sequences When Using Polymerase Chain Reaction-Based Diagnosis for Rett Syndrome

5. Allelic Dropout Can Cause False-Positive Results for Prader-Willi and Angelman Syndrome Testing

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. G-quadruplexes and associated proteins in aging and Alzheimer’s disease;Frontiers in Aging;2023-06-01

2. The Dynamic Regulation of G-Quadruplex DNA Structures by Cytosine Methylation;International Journal of Molecular Sciences;2022-02-22

3. False negatives in GBA1 sequencing due to polymerase dependent allelic imbalance;Scientific Reports;2021-01-08

4. DNA G‐Quadruplexes (G4s) Modulate Epigenetic (Re)Programming and Chromatin Remodeling;BioEssays;2019-08-05

5. Hypothesis: Regulation of neuroplasticity may involve I-motif and G-quadruplex DNA formation modulated by epigenetic mechanisms;Medical Hypotheses;2019-06

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