Replication Bypass of the <i>N</i>-(2-Deoxy-<scp>d</scp>-erythro-pentofuranosyl)-urea DNA Lesion by Human DNA Polymerase η-Reference-Cited by-同舟云学术

Replication Bypass of the N-(2-Deoxy-d-erythro-pentofuranosyl)-urea DNA Lesion by Human DNA Polymerase η

Published:2024-02-27 Issue:6 Volume:63 Page:754-766
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Tomar Rachana¹,Li Songlin¹,Egli Martin²^ORCID,Stone Michael P.¹^ORCID

Affiliation:

1. Department of Chemistry, Vanderbilt Ingram Cancer Center, and Vanderbilt Center for Structural Biology, Vanderbilt University, Nashville, Tennessee 37235, United States

2. Department of Biochemistry, School of Medicine, Vanderbilt Ingram Cancer Center, and Vanderbilt Center for Structural Biology, Vanderbilt University, Nashville, Tennessee 37232, United States

Funder

U.S. Department of Health and Human Services

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.3c00569

Reference46 articles.

1. Thymine glycols and urea residues in M13 DNA constitute replicative blocksin vitro

2. Thymine ring saturation and fragmentation products: lesion bypass, misinsertion and implications for mutagenesis

3. Replication Inhibition and Miscoding Properties of DNA Templates Containing a Site-Specific cis-Thymine Glycol or Urea Residue

4. Urea Lesion Formation in DNA as a Consequence of 7,8-Dihydro-8-oxoguanine Oxidation and Hydrolysis Provides a Potent Source of Point Mutations

5. Insertion of the fragile 2′-deoxyribosylurea residue into oligodeoxynucleotides

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