Probing Enhanced Double-Strand Break Formation at Abasic Sites within Clustered Lesions in Nucleosome Core Particles
Author:
Affiliation:
1. Department of Chemistry, Johns Hopkins University, Baltimore, Maryland 21218, United States
Funder
National Institute of General Medical Sciences
Publisher
American Chemical Society (ACS)
Subject
Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.6b01144
Reference55 articles.
1. Non-homologous end joining: Emerging themes and unanswered questions
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5. An Inhibitor of Nonhomologous End-Joining Abrogates Double-Strand Break Repair and Impedes Cancer Progression
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