Modification of Keap1 Cysteine Residues by Sulforaphane
Author:
Affiliation:
1. Department of Medicinal Chemistry and Pharmacognosy, and ‡Center for Pharmaceutical BiotechnologyUniversity of Illinois College of Pharmacy, Chicago, Illinois 60612, United States
Publisher
American Chemical Society (ACS)
Subject
Toxicology,General Medicine
Link
https://pubs.acs.org/doi/pdf/10.1021/tx100389r
Reference35 articles.
1. Keap1 Is a Redox-Regulated Substrate Adaptor Protein for a Cul3-Dependent Ubiquitin Ligase Complex
2. Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain
3. Molecular Mechanisms Activating the Nrf2-Keap1 Pathway of Antioxidant Gene Regulation
4. Identification of a common chemical signal regulating the induction of enzymes that protect against chemical carcinogenesis.
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