Hi1a Improves Sensorimotor Deficit following Endothelin-1-Induced Stroke in Rats but Does Not Improve Functional Outcomes following Filament-Induced Stroke in Mice-Reference-Cited by-同舟云学术

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Hi1a Improves Sensorimotor Deficit following Endothelin-1-Induced Stroke in Rats but Does Not Improve Functional Outcomes following Filament-Induced Stroke in Mice

Published:2024-03-14 Issue:4 Volume:7 Page:1043-1054
ISSN:2575-9108
Container-title:ACS Pharmacology & Translational Science
language:en
Short-container-title:ACS Pharmacol. Transl. Sci.

Author:

Knezic Adriana¹^ORCID,Budusan Elena²,Saez Natalie J.³⁴,Broughton Brad R. S.¹,Rash Lachlan D.²^ORCID,King Glenn F.³⁴,Widdop Robert E.¹,McCarthy Claudia A.¹

Affiliation:

1. Cardiovascular Disease Program, Monash Biomedicine Discovery Institute (BDI), Department of Pharmacology, Monash University, Clayton, VIC 3800, Australia

2. School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, St Lucia, QLD 4072, Australia

3. Institute for Molecular Bioscience, The University of Queensland, St Lucia, QLD 4072, Australia

4. Australian Research Council Centre of Excellence for Innovations in Peptide and Protein Science, The University of Queensland, St Lucia, QLD 4072, Australia

Funder

National Health and Medical Research Council

Department of Education, Australian Government

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acsptsci.3c00328

Reference86 articles.

1. Thrombolysis for acute ischaemic stroke: current status and future perspectives

2. N-Methyl-D-Aspartate antagonist reduces stroke size and regional glucose metabolism

3. Selfotel in Acute Ischemic Stroke

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