Pharmacologic Targeting of the C-Terminus of Heat Shock Protein 90 Improves Neuromuscular Function in Animal Models of Charcot Marie Tooth X1 Disease-Reference-Cited by-同舟云学术

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Pharmacologic Targeting of the C-Terminus of Heat Shock Protein 90 Improves Neuromuscular Function in Animal Models of Charcot Marie Tooth X1 Disease

Published:2023-01-20 Issue:2 Volume:6 Page:306-319
ISSN:2575-9108
Container-title:ACS Pharmacology & Translational Science
language:en
Short-container-title:ACS Pharmacol. Transl. Sci.

Author:

Kaur Sukhmanjit¹,Zhang Xinyue¹^ORCID,Patel Sugandha¹,Rodriguez Yssa A.¹,Luther Kylie J.¹,Alghafli Ghufran¹,Lang Ryan M.¹,Abrams Charles K.²,Dobrowsky Rick T.¹^ORCID

Affiliation:

1. Department of Pharmacology and Toxicology, University of Kansas, Lawrence, Kansas 66045, United States

2. Department of Neurology and Rehabilitation and Biomedical Engineering, College of Medicine, University of Illinois Chicago, Chicago, Illinois 60612, United States

Funder

National Institute of Neurological Disorders and Stroke

Publisher

American Chemical Society (ACS)

Subject

Pharmacology (medical),Pharmacology

Link

https://pubs.acs.org/doi/pdf/10.1021/acsptsci.2c00223

Reference62 articles.

1. GJB1-associated X-linked Charcot–Marie–Tooth disease, a disorder affecting the central and peripheral nervous systems

2. X-linked Charcot-Marie-Tooth disease

3. Molecular mechanisms of inherited demyelinating neuropathies

4. What’s the Function of Connexin 32 in the Peripheral Nervous System?

5. Diverse Trafficking Abnormalities of Connexin32 Mutants Causing CMTX

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