If You Cannot Win Them, Join Them: Understanding New Ways to Target STAT3 by Small Molecules
Author:
Affiliation:
1. Chemistry, School of Natural and Environmental Sciences, Newcastle University, NE1 7RU Newcastle, United Kingdom
2. IQS School of Engineering(IQS)—Universitat Ramon Llull (URL), 08017 Barcelona, Spain
Funder
School of Natural and Environmental Sciences, Newcastle University
Publisher
American Chemical Society (ACS)
Subject
General Chemical Engineering,General Chemistry
Link
http://pubs.acs.org/doi/pdf/10.1021/acsomega.9b01601
Reference52 articles.
1. Structural Biology of STAT3 and Its Implications for Anticancer Therapies Development
2. Resistance to chemotherapy via Stat3-dependent overexpression of Bcl-2 in metastatic breast cancer cells
3. A new role for STAT3 as a regulator of chromatin topology
4. Mitochondrial dysfunction induced by a SH2 domain-targeting STAT3 inhibitor leads to metabolic synthetic lethality in cancer cells
5. Drug Resistance via Feedback Activation of Stat3 in Oncogene-Addicted Cancer Cells
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