Physiologically Based Pharmacokinetic Model To Predict Metoprolol Disposition in Healthy and Disease Populations-Reference-Cited by-同舟云学术

Physiologically Based Pharmacokinetic Model To Predict Metoprolol Disposition in Healthy and Disease Populations

Published:2023-08-03 Issue:32 Volume:8 Page:29302-29313
ISSN:2470-1343
Container-title:ACS Omega
language:en
Short-container-title:ACS Omega

Author:

Zamir Ammara¹,Rasool Muhammad Fawad¹^ORCID,Imran Imran²^ORCID,Saeed Hamid³,Khalid Sundus¹,Majeed Abdul¹,Rehman Anees Ur¹,Ahmad Tanveer⁴,Alasmari Fawaz⁵,Alqahtani Faleh⁵

Affiliation:

1. Department of Pharmacy Practice, Faculty of Pharmacy, Bahauddin Zakariya University, Multan 60800, Pakistan

2. Department of Pharmacology, Faculty of Pharmacy, Bahauddin Zakariya University, Multan 60800, Pakistan

3. Section of Pharmaceutics, University College of Pharmacy, University of the Punjab, Allama Iqbal Campus, Lahore 54000, Pakistan

4. Institute for Advanced Biosciences (IAB), CNRS UMR5309, INSERM U1209, Grenoble Alpes University, La Tronche 38700, France

5. Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia

Funder

King Saud University

Publisher

American Chemical Society (ACS)

Subject

General Chemical Engineering,General Chemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acsomega.3c02673

Reference51 articles.

1. PBPK modeling and simulation in drug research and development

2. Development and evaluation of physiologically based pharmacokinetic drug-disease models for predicting captopril pharmacokinetics in chronic diseases

3. Physiologically-Based Pharmacokinetics in Drug Development and Regulatory Science

4. Applications of linking PBPK and PD models to predict the impact of genotypic variability, formulation differences, differences in target binding capacity and target site drug concentrations on drug responses and variability

5. A Physiologically Based Pharmacokinetic Drug–Disease Model to Predict Carvedilol Exposure in Adult and Paediatric Heart Failure Patients by Incorporating Pathophysiological Changes in Hepatic and Renal Blood Flows

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