Endoplasmic Reticulum Stress-Activated PERK-eIF2α-ATF4 Signaling Pathway is Involved in the Ameliorative Effects of Ginseng Polysaccharides against Cisplatin-Induced Nephrotoxicity in Mice-Reference-Cited by-同舟云学术

Endoplasmic Reticulum Stress-Activated PERK-eIF2α-ATF4 Signaling Pathway is Involved in the Ameliorative Effects of Ginseng Polysaccharides against Cisplatin-Induced Nephrotoxicity in Mice

Published:2021-03-24 Issue:13 Volume:6 Page:8958-8966
ISSN:2470-1343
Container-title:ACS Omega
language:en
Short-container-title:ACS Omega

Author:

Wei Xiao-meng¹,Jiang Shuang¹²,Li Shan-shan³,Sun Yin-shi³,Wang Shi-han¹²,Liu Wen-cong¹²,Wang Zi¹²,Wang Ying-ping¹²,Zhang Rui¹²,Li Wei¹²^ORCID

Affiliation:

1. College of Chinese Medicinal Materials, Jilin Agricultural University, Changchun 130118, China

2. National & Local Joint Engineering Research Center for Ginseng Breeding and Development, Changchun 130118, China

3. Institute of Special Wild Economic Animals and Plant, Chinese Academy of Agricultural Sciences, Changchun 132109, China

Funder

People's Government of Jilin Province

Publisher

American Chemical Society (ACS)

Subject

General Chemical Engineering,General Chemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acsomega.0c06339

Reference43 articles.

1. Overview of cisplatin-induced neurotoxicity and ototoxicity, and the protective agents

2. Vagus nerve stimulation mediates protection from kidney ischemia-reperfusion injury through α7nAChR+ splenocytes

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4. CaSR activates PKCδ to induce cardiomyocyte apoptosis via ER stress‑associated apoptotic pathways during ischemia/reperfusion

5. Toll-like Receptor 4-mediated Endoplasmic Reticulum Stress in Intestinal Crypts Induces Necrotizing Enterocolitis

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