Glutamyl Substrate-Induced Exposure of a Free Cysteine Residue in the Vitamin K-Dependent γ-Glutamyl Carboxylase Is Critical for Vitamin K Epoxidation-Reference-Cited by-同舟云学术

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Glutamyl Substrate-Induced Exposure of a Free Cysteine Residue in the Vitamin K-Dependent γ-Glutamyl Carboxylase Is Critical for Vitamin K Epoxidation

Published:1999-06-26 Issue:29 Volume:38 Page:9517-9523
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Bouchard Beth A.¹,Furie Bruce¹,Furie Barbara C.¹

Affiliation:

1. Center for Hemostasis & Thrombosis Research, Division of Hematology−Oncology, Research East 319, Beth Israel Deaconess Medical Center, P.O. Box 15732, Boston, Massachusetts 02215, and Department of Medicine, Harvard Medical School, Boston, Massachusetts

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi9907375

Reference39 articles.

1. Differentiation of metal ion-induced transitions of prothrombin fragment 1.

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3. Effect of Vitamin K-dependent Protein Precursor Propeptide, Vitamin K Hydroquinone, and Glutamate Substrate Binding on the Structure and Function of γ-Glutamyl Carboxylase;Journal of Biological Chemistry;2010-10

4. Electrophilicity of quinones and its relationship with hydride affinity;Chemical Physics Letters;2009-03

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