Controlling Amyloid Beta Peptide Aggregation and Toxicity by Protease-Stable Ligands-Reference-Cited by-同舟云学术

Controlling Amyloid Beta Peptide Aggregation and Toxicity by Protease-Stable Ligands

Published:2023-02-15 Issue:2 Volume:3 Page:158-173
ISSN:2694-2437
Container-title:ACS Bio & Med Chem Au
language:en
Short-container-title:ACS Bio Med Chem Au

Author:

Mallesh Rathnam¹²³^ORCID,Juhee khan ¹²^ORCID,Gharai Prabir Kumar¹²^ORCID,Gupta Varsha¹²,Roy Rajsekhar¹,Ghosh Surajit¹²³^ORCID

Affiliation:

1. Department of Bioscience & Bioengineering, Indian Institute of Technology, Jodhpur, NH 65, Surpura Bypass Road, Karwar, Rajasthan 342037, India

2. Organic and Medicinal Chemistry and Structural Biology and Bioinformatics Division, CSIR-Indian Institute of Chemical Biology, 4, Raja S. C. Mullick Road, Jadavpur, Kolkata, West Bengal 700 032, India

3. National Institute of Pharmaceutical Education and Research, Kolkata, Chunilal Bhawan 168, Maniktala Main Road, Kolkata 700054, India

Funder

Science and Engineering Research Board

Publisher

American Chemical Society (ACS)

Subject

Pharmaceutical Science,Drug Discovery,Molecular Biology,Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acsbiomedchemau.2c00067

Reference35 articles.

1. Neuritic plaques and cerebrovascular amyloid in Alzheimer disease are antigenically related.

2. Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein

3. beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease.

4. Amyloid β-Protein and the Genetics of Alzheimer's Disease

5. Self-association of β-Amyloid Peptide (1–40) in Solution and Binding to Lipid Membranes

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