Reactive Oxygen Species-Activated Akt/ASK1/p38 Signaling Pathway in Nickel Compound-Induced Apoptosis in BEAS 2B Cells-Reference-Cited by-同舟云学术

Reactive Oxygen Species-Activated Akt/ASK1/p38 Signaling Pathway in Nickel Compound-Induced Apoptosis in BEAS 2B Cells

Published:2010-01-29 Issue:3 Volume:23 Page:568-577
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Pan Jingju¹,Chang Qingshan¹,Wang Xin¹,Son Youngok¹,Zhang Zhuo¹,Chen Gang¹,Luo Jia¹,Bi Yongyi¹,Chen Fei¹,Shi Xianglin¹

Affiliation:

1. Department of Occupational and Environmental Health, School of Public Health, Wuhan University, Wuhan 430071, China, Graduate Center for Toxicology and Department of Medicine, University of Kentucky, 234 Health Science Research Building, Lexington, Kentucky 40536, and Laboratory of Cancer Signaling and Epigenetics, Pathology and Physiology Research Branch, Health Effects Laboratory Division, National Institute for Occupational Safety and Health, 1095 Willowdale Road, Morgantown, West Virginia 26505

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/tx9003193

Reference66 articles.

1. Molecular Mechanisms of Nickel Carcinogenesis

2. Cancers of the Lung and Nasal Sinuses in Nickel Workers

3. Metal-induced apoptosis: mechanisms

4. Toxic effect of nickel in an in vitro model of human oral epithelium

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