The G Protein-Coupled Receptor GPR4 Suppresses ERK Activation in a Ligand-Independent Manner-Reference-Cited by-同舟云学术

The G Protein-Coupled Receptor GPR4 Suppresses ERK Activation in a Ligand-Independent Manner

Published:2003-10-01 Issue:42 Volume:42 Page:12181-12191
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Bektas Meryem¹,Barak Larry S.¹,Jolly Puneet S.¹,Liu Hong¹,Lynch Kevin R.¹,Lacana Emanuela¹,Suhr Ki-Beom¹,Milstien Sheldon¹,Spiegel Sarah¹

Affiliation:

1. Department of Biochemistry, Virginia Commonwealth University School of Medicine, Richmond, Virginia 23298, Department of Cell Biology, Duke University Medical Center, Durham, North Carolina 27710, Department of Pharmacology, University of Virginia School of Medicine, Charlottesville, Virginia 22908, Laboratory of Immunology, Food and Drug Administration, Bethesda, Maryland 20892, Department of Dermatology, Chungnam National University School of Medicine, Daesa-dong 640, Jung-ku, Taejon, 301040 Korea, and...

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi035051y

Reference71 articles.

1. Diversity of cellular receptors and functions for the lysophospholipid growth factors lysophosphatidic acid and sphingosine 1‐phosphate

2. Sphingosylphosphocholine is a naturally occurring lipid mediator in blood plasma: a possible role in regulating cardiac function via sphingolipid receptors

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