Properties of Human Vasopressin Precursor Constructs: Inefficient Monomer Folding in the Absence of Copeptin as a Potential Contributor to Diabetes Insipidus-Reference-Cited by-同舟云学术

Properties of Human Vasopressin Precursor Constructs: Inefficient Monomer Folding in the Absence of Copeptin as a Potential Contributor to Diabetes Insipidus

Published:2004-06-01 Issue:25 Volume:43 Page:8191-8203
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Barat Chandana¹,Simpson LeRone¹,Breslow Esther¹

Affiliation:

1. Department of Biochemistry, Weill Medical College of Cornell University, 1300 York Avenue, New York, New York 10021

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi0400094

Reference43 articles.

1. A diabetes insipidus vasopressin prohormone altered outside the central core of neurophysin accumulates in the endoplasmic reticulum

2. Mechanism of Endoplasmic Reticulum Retention of Mutant Vasopressin Precursor Caused by a Signal Peptide Truncation Associated with Diabetes Insipidus

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