Electrophilic Components of Diesel Exhaust Particles (DEP) Activate Transient Receptor Potential Ankyrin-1 (TRPA1): A Probable Mechanism of Acute Pulmonary Toxicity for DEP-Reference-Cited by-同舟云学术

Electrophilic Components of Diesel Exhaust Particles (DEP) Activate Transient Receptor Potential Ankyrin-1 (TRPA1): A Probable Mechanism of Acute Pulmonary Toxicity for DEP

Published:2011-06-20 Issue:6 Volume:24 Page:950-959
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Deering-Rice Cassandra E.,Romero Erin G.,Shapiro Darien,Hughen Ronald W.,Light Alan R.,Yost Garold S.,Veranth John M.,Reilly Christopher A.

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/tx200123z

Reference63 articles.

1. Sources of ambient fine particulate matter at two community sites in Detroit, Michigan

2. Current Status of the Toxicology of Diesel Engine Exhaust — and the ACES Project

3. TRPA1 and Sympathetic Activation Contribute to Increased Risk of Triggered Cardiac Arrhythmias in Hypertensive Rats Exposed to Diesel Exhaust

4. Expression and function of the ion channel TRPA1 in vagal afferent nerves innervating mouse lungs

5. Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c-fibers and colocalization with trk receptors

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