Maltol Mitigates Thioacetamide-induced Liver Fibrosis through TGF-β1-mediated Activation of PI3K/Akt Signaling Pathway-Reference-Cited by-同舟云学术

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Maltol Mitigates Thioacetamide-induced Liver Fibrosis through TGF-β1-mediated Activation of PI3K/Akt Signaling Pathway

Published:2019-01-15 Issue:5 Volume:67 Page:1392-1401
ISSN:0021-8561
Container-title:Journal of Agricultural and Food Chemistry
language:en
Short-container-title:J. Agric. Food Chem.

Author:

Mi Xiao-jie¹,Hou Jin-gang¹²,Jiang Shuang¹,Liu Zhi¹,Tang Shan¹,Liu Xiang-xiang¹,Wang Ying-ping¹³,Chen Chen⁴,Wang Zi¹,Li Wei¹³^ORCID

Affiliation:

1. College of Chinese Medicinal Materials, Jilin Agricultural University, Changchun 130118, China

2. Intelligent Synthetic Biology Center, Daejeon 34141, Republic of Korea

3. National & Local Joint Engineering Research Center for Ginseng Breeding and Development, Changchun 130118, China

4. School of Biomedical Sciences, Queensland Brain Institute, The University of Queensland, Brisbane, Queensland 4072, Australia

Funder

Natural Science Foundation of Jilin Province

People's Government of Jilin Province

National Natural Science Foundation of China

Jilin Agricultural University

Publisher

American Chemical Society (ACS)

Subject

General Agricultural and Biological Sciences,General Chemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.jafc.8b05943

Reference52 articles.

1. Liver fibrogenesis and the role of hepatic stellate cells: New insights and prospects for therapy

2. Liver cirrhosis

3. Targeting liver fibrosis: Strategies for development and validation of antifibrotic therapies

4. Role of CYP2E1 in thioacetamide-induced mouse hepatotoxicity

5. A bioinformatic and mechanistic study elicits the antifibrotic effect of ursolic acid through the attenuation of oxidative stress with the involvement of ERK, PI3K/Akt, and p38 MAPK signaling pathways in human hepatic stellate cells and rat liver

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