Abstract
The question whether asphyxia, acute cerebral anæmia and sensory stimuli are accompanied by an increased liberation of adrenaline from the suprarenal glands has been vigorously debated for the last ten years. It would require a monograph to give an adequate review of the literature and of the experiments bearing on this subject. This has been done to some extent in the latest papers by Kodama (1). The great majority of observers find under these conditions an increased concentration of adrenaline in the general circulation, so far as tested by means of different denervated organs. Since this increase in concentration was found to depend on the integrity of the splanchnic nerves it was concluded that the secretory fibres of the splanchnic nerves were responsible for this increase and that the increase itself is an expression of an augmented liberation of adrenaline from the suprarenal glands.
Reference23 articles.
1. Kodama ` Tohoku J. Exper. Med. ' vol. 4 pp. 166 465 601 (1923-24).
2. Gley and Quinquaud ' C.R. Soc. Biol. ' vol. 80 p. 15 (1917)
3. ' J. Physiol et Pathol. gen. ' vol. 17 p. 807 (1918)
4. ' Arch intern. Physiol. ' vol. 18 p. 22 (1921).
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