Huntington disease: Advances in molecular and cell biology
Author:
Affiliation:
1. ; Institute of Medical Genetics; University of Wales College of Medicine; Heath Park Cardiff CF4 4XN UK
Publisher
Wiley
Subject
Genetics (clinical),Genetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1023/A:1005340302695/fullpdf
Reference71 articles.
1. Structure and expression of the Huntington's disease gene: evidence against simple inactivation due to an expanded CAG repeat;Ambrose;Somat Cell Mol Genet,1994
2. The relationship between trinucleotide (CAG) repeat length and clinical features of Huntington's disease;Andrew;Nature Genet,1993
3. Defined neurofilament, τ, and β-amyloid precursor protein epitopes distinguish Alzheimer from non-Alzheimer senile plaques;Arai;Proc Natl Acad Sci USA,1990
4. CAG expansion affects the expression of mutant huntingtin in the Huntington's disease brain;Aronin;Neuron,1995
5. Cloning of the essential myotonic dystrophy region and mapping of the putative defect;Aslanidis;Nature,1992
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