Hypereosinophilia: An Unusual Bystander in Cancer
Author:
Affiliation:
1. Section of Benign Hematology, Department on Pulmonary Medicine, The University of Texas MD Anderson Cancer Center, Houston, TX, USA
Publisher
Innovative Healthcare Institute
Subject
Cancer Research,Oncology,Immunology,Immunology and Allergy
Link
https://meridian.allenpress.com/innovationsjournals-JIPO/article-pdf/4/1/26/3010568/i2590-017x-4-1-26.pdf
Reference10 articles.
1. McNagny K, Graf T. Making eosinophils through subtle shifts in transcription factor expression. J Exp Med. 2002; 195: F43– F47.
2. Rosenberg HF, Dyer KD, Foster PS. Eosinophils: changing perspectives in health and disease. Nat Rev Immunol. 2013; 13: 9– 22.
3. Melo RC, Spencer LA, Perez SA, et al. Human eosinophils secrete preformed, granule-stored interleukin-4 through distinct vesicular compartments. Traffic. 2005; 6: 1047– 1057.
4. Roufosse F, De Lavareille A, Schandené L, et al. Mepolizumab as a corticosteroid-sparing agent in lymphocytic variant hypereosinophilic syndrome. J Allergy Clin Immunol. 2010; 126: 828– 835.e3.
5. Cools J, DeAngelo DJ, Gotlib J, et al. A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome. N Engl J Med. 2003; 348: 1201– 1214.
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